An Attempt at Explaining Null-Hypothesis Testing and Statistical Power with 1 Figure and 1,500 Words

Is a Figure worth 1,500 words?



Significance Testing

1. The red curve shows the sampling distribution if there is no effect. Most results will give a signal/noise ratio close to 0 because there is no effect (0/1 = 0)

2. Sometimes sampling error can produce large signals, but these events are rare

3. To be sure that we have a real signal, we can chose a high criterion to decide that there was an effect (reject H0). Normally, we use a 2:1 ratio (z > 2) to do so, but we could use a higher or lower criterion value.  This value is shown by the green vertical line in the Figure

4. z-score greater than 2 leaves only 2.5% of the red distribution. This means we would expect only 2.5% of outcomes with z-scores greater than 2 if there is no effect. If we would use the same criterion for negative effects, we would get another 2.5% in the lower tail of the red distribution. Combined we would have 5% of cases where we have a false positive, that is, we decide that there is an effect when there was no effect. This is why we say, p < .05 to call a result significant. The probabilty (p) of a false positive result is no greater than 5% if we keep on repeating studies and using z > 2 as the criterion to claim an effect. If there is never an effect in any of the studies we are doing, we end up with 5% false positive results. A false positive is also called a type-I error. We are making the mistake to infer from our study that an effect is present when there is no effect.

Statistical Power

5. Now that you understand significance testing (LOL), we can introduce the concept of statistical power. Effects can be large or small. For example, gender differences in height are large, gender differences in the number of sexual partners are small.  Also studies can have a lot of sampling error or very little sampling error.  A study of 10 men and 10 women may accidentally include 2 women who are on the basketball team.  A study of 1000 men and women is likely to be more representative of the population.  Based on the effect size in the population and sample size, the true signal (effect size in the population) to noise (sampling error) ratio can differ.  The higher the signal to noise ratio is, the further away the sampling distribution of the real data (the blue curve) will be.  In the figure below the population effect size and sampling error produced a z-score of 2.8, but actual samples will never produce this value. Sampling error will again produce different z-scores above or below the expected value of 2.8.  Most samples will produce values close to 2.8, but some samples will produce more extreme deviations.  Samples that overestimate the expected value of 2.8 are not a problem because these values are all greater than the criterion for statistical significance. So, in all of these samples we will make the right decision to infer that an effect is present when an effect is present. A so called true positive result.  Even if sampling error leads to a small underestimation of the expected value of 2.8, the values can still be above the criterion for statistical significance and we get a true positive result.

6. When sampling error leads to more extreme underestimation of the expected value of 2.8, samples may produce results with a z-score less than 2.  Now the result is no longer statistically significant. These cases are called false negatives or type-II errors.  We fail to infer that an effect is present, when there actually is an effect (think about a faulty pregnancy test that fails to detect that a woman is pregnant).  It does not matter whether we actually infer that there is no effect or remain indecisive about the presence of an effect. We did a study where an effect exists and we failed to provide sufficient evidence for it.

7. The Figure shows the probability of making a type-II error as the area of the blue curve on the left side of the green line.  In this example, 20% of the blue curve is on the left side of the green line. This means 20% of all samples with an expected value of 2.8 will produce false negative results.

8. We can also focus on the area of the blue curve on the right side of the green line.  If 20% of the area is on the left side, 80% of the area must be on the right side.  This means, we have an 80% probability to obtain a true positive result; that is, a statistically significant result where the observed z-score is greater than the criterion z-score of 2.   This probability is called statistical power.  A study with high power has a high probability to discover real effects by producing z-scores greater than the criterion value. A study with low power has a high probability to produce a false negative result by producing z-scores below the criterion value.

9. Power depends on the criterion value and the expected value.  We could reduce the type-II error and increase power in the Figure by moving the green line to the left.  As we reduce the criterion to claim an effect, we reduce the area of the blue curve on the left side of the line. We are now less likely to encounter false negative results when an effect is present.  However, there is a catch.  By moving the green line to the left, we are increasing the area of the red curve on the right side of the red curve. This means, we are increasing the probability of a false positive result.  To avoid this problem we can keep the green line where it is and move the expected value of the blue line to the right.  By shifting the blue curve to the right, a smaller area of the blue curve will be on the left side of green line.

10. In order to move the blue curve to the right we need to increase the effect size or reduce sampling error.  In experiments it may be possible to use more powerful manipulations to increase effect sizes.  However, often increasing effect sizes is not an option.  How would you increase the effect size of sex on sexual partners?  Therefore, your best option is to reduce sampling error.  As sampling error decreases, the blue curve moves further to the right and statistical power increases.

Practical Relevance: The Hunger Games of Science: With high power the odds are always in your favor

10. Learning about statistical power is important because the outcome of your studies does not just depend on your expertise. It also depends on factors that are not under your control. Sampling error can sometimes help you to get significance by giving you z-scores higher than the expected value, but these z-scores will not replicate because sampling error can also be your enemy and lower your z-scores.  In this way, each study that you do is a bit like playing the lottery or a box of chocolates. You never know how much sampling error you will get.  The good news is that you are in charge of the number of winning tickets in the lottery.  A study with 20% power, has only 20% winning tickets.  The other 80% say, “please play again.”  A study with 80% power has 80% winning tickets.  You have a high chance to get a significant result and you or others will be able to redo the study and again have a high chance to replicate your original result.  It can be embarrassing when somebody conducts a replication study of your significant result and ends up with a failure to replicate your finding.  You can avoid this outcome by conducting studies with high statistical power.

11. Of course, there is a price to pay. Reducing sampling error often requires more time and participants. Unfortunately, the costs increase exponentially.  It is easier to increase statistical power from 20% to 50% than to increase it from 50% to 80%. It is even more costly to increase it from 80% to 90%.  This is what economists call diminishing marginal utility.  Initially you get a lot of bang for your buck, but eventually the costs for any real gains are too high.  For this reason, Cohen (1988) recommended that researchers should aim for 80% power in their studies.  This means that 80% of your initial attempts to demonstrate an effect will succeed when your hard work in planning and conducting a study produced a real effect.  For 20% of the study you may either give up or try again to see whether your fist study produced a true negative result (there is no effect) or a false negative result (you did everything correctly, but sampling error handed you a losing ticket.  Failure is part of life, but you have some control over the amount of failures that you encounter.

12. The End. You are now ready to learn how you can conduct power analysis for actual studies to take control your fate.  Be a winner, not a loser.


Random measurement error and the replication crisis: A statistical analysis

This is a draft of a commentary on Loken and Gelman’s Science article “Measurement error and the replication crisis. Comments are welcome.

Random Measurement Error Reduces Power, Replicability, and Observed Effect Sizes After Selection for Significance

Ulrich Schimmack and Rickard Carlsson

In the article “Measurement error and the replication crisis” Loken and Gelman (LG) “caution against the fallacy of assuming that that which does not kill statistical significance makes it stronger” (1). We agree with the overall message that it is a fallacy to interpret observed effect size estimates in small samples as accurate estimates of population effect sizes.  We think it is helpful to recognize the key role of statistical power in significance testing.  If studies have less than 50% power, effect sizes must be inflated to be significant. Thus, all observed effect sizes in these studies are inflated.  Once power is greater than 50%, it is possible to obtain significance with observed effect sizes that underestimate the population effect size. However, even with 80% power, the probability of overestimation is 62.5%. [corrected]. As studies with small samples and small effect sizes often have less than 50% power (2), we can safely assume that observed effect sizes overestimate the population effect size. The best way to make claims about effect sizes in small samples is to avoid interpreting the point estimate and to interpret the 95% confidence interval. It will often show that significant large effect sizes in small samples have wide confidence intervals that also include values close to zero, which shows that any strong claims about effect sizes in small samples are a fallacy (3).

Although we agree with Loken and Gelman’s general message, we believe that their article may have created some confusion about the effect of random measurement error in small samples with small effect sizes when they wrote “In a low-noise setting, the theoretical results of Hausman and others correctly show that measurement error will attenuate coefficient estimates. But we can demonstrate with a simple exercise that the opposite occurs in the presence of high noise and selection on statistical significance” (p. 584).  We both read this sentence as suggesting that under the specified conditions random error may produce even more inflated estimates than perfectly reliable measure. We show that this interpretation of their sentence would be incorrect and that random measurement error always leads to an underestimation of observed effect sizes, even if effect sizes are selected for significance. We demonstrate this fact with a simple equation that shows that true power before selection for significance is monotonically related to observed power after selection for significance. As random measurement error always attenuates population effect sizes, the monotonic relationship implies that observed effect sizes with unreliable measures are also always attenuated.  We provide the formula and R-Code in a Supplement. Here we just give a brief description of the steps that are involved in predicting the effect of measurement error on observed effect sizes after selection for significance.

The effect of random measurement error on population effect sizes is well known. Random measurement error adds variance to the observed measures X and Y, which lowers the observable correlation between two measures. Random error also increases the sampling error. As the non-central t-value is the proportion of these two parameters, it follows that random measurement error always attenuates power. Without selection for significance, median observed effect sizes are unbiased estimates of population effect sizes and median observed power matches true power (4,5). However, with selection for significance, non-significant results with low observed power estimates are excluded and median observed power is inflated. The amount of inflation is proportional to true power. With high power, most results are significant and inflation is small. With low power, most results are non-significant and inflation is large.


Schimmack developed a formula that specifies the relationship between true power and median observed power after selection for significance (6). Figure 1 shows that median observed power after selection for significant is a monotonic function of true power.  It is straightforward to transform inflated median observed power into median observed effect sizes.  We applied this approach to Locken and Gelman’s simulation with a true population correlation of r = .15. We changed the range of sample sizes from 50 to 3050 to 25 to 1000 because this range provides a better picture of the effect of small samples on the results. We also increased the range of reliabilities to show that the results hold across a wide range of reliabilities. Figure 2 shows that random error always attenuates observed effect sizes, even after selection for significance in small samples. However, the effect is non-linear and in small samples with small effects, observed effect sizes are nearly identical for different levels of unreliability. The reason is that in studies with low power, most of the observed effect is driven by the noise in the data and it is irrelevant whether the noise is due to measurement error or unexplained reliable variance.


In conclusion, we believe that our commentary clarifies how random measurement error contributes to the replication crisis.  Consistent with classic test theory, random measurement error always attenuates population effect sizes. This reduces statistical power to obtain significant results. These non-significant results typically remain unreported. The selective reporting of significant results leads to the publication of inflated effect size estimates. It would be a fallacy to consider these effect size estimates reliable and unbiased estimates of population effect sizes and to expect that an exact replication study would also produce a significant result.  The reason is that replicability is determined by true power and observed power is systematically inflated by selection for significance.  Our commentary also provides researchers with a tool to correct for the inflation by selection for significance. The function in Figure 1 can be used to deflate observed effect sizes. These deflated observed effect sizes provide more realistic estimates of population effect sizes when selection bias is present. The same approach can also be used to correct effect size estimates in meta-analyses (7).


1. Loken, E., & Gelman, A. (2017). Measurement error and the replication crisis. Science,

355 (6325), 584-585. [doi: 10.1126/science.aal3618]

2. Cohen, J. (1962). The statistical power of abnormal-social psychological research: A review. Journal of Abnormal and Social Psychology, 65, 145-153,

3. Cohen, J. (1994). The earth is round (p < .05). American Psychologist, 49, 997-1003.

4. Schimmack, U. (2012). The ironic effect of significant results on the credibility of multiple-study articles. Psychological Methods, 17(4), 551-566.

5. Schimmack, U. (2016). A revised introduction to the R-Index.

6. Schimmack, U. (2017). How selection for significance influences observed power.

7. van Assen, M.A., van Aert, R.C., Wicherts, J.M. (2015). Meta-analysis using effect size distributions of only statistically significant studies. Psychological Methods, 293-309. doi: 10.1037/met0000025.


#### R-CODE ###


### sample sizes

N = seq(25,500,5)

### true population correlation

true.pop.r = .15

### reliability

rel = 1-seq(0,.9,.20)

### create matrix of population correlations between measures X and Y.

obs.pop.r = matrix(rep(true.pop.r*rel),length(N),length(rel),byrow=TRUE)

### create a matching matrix of sample sizes

N = matrix(rep(N),length(N),length(rel))

### compute non-central t-values

ncp.t = obs.pop.r / ( (1-obs.pop.r^2)/(sqrt(N – 2)))

### compute true power

true.power = pt(ncp.t,N-2,qt(.975,N-2))

###  Get Inflated Observed Power After Selection for Significance

inf.obs.pow = pnorm(qnorm(true.power/2+(1-true.power),qnorm(true.power,qnorm(.975))),qnorm(.975))

### Transform Into Inflated Observed t-values

inf.obs.t = qt(inf.obs.pow,N-2,qt(.975,N-2))

### Transform inflated observed t-values into inflated observed effect sizes = (sqrt(N + 4*inf.obs.t^2 -2) – sqrt(N – 2))/(2*inf.obs.t)

### Set parameters for Figure

x.min = 0

x.max = 500

y.min = 0.10

y.max = 0.45

ylab = “Inflated Observed Effect Size”

title = “Effect of Selection for Significance on Observed Effect Size”

### Create Figure

for (i in 1:length(rel)) {


plot(N[,1],[,i],type=”l”,xlim=c(x.min,x.max),ylim=c(y.min,y.max),col=col[i],xlab=”Sample Size”,ylab=”Median Observed Effect Size After Selection for Significance”,lwd=3,main=title)

segments(x0 = 600,y0 = y.max-.05-i*.02, x1 = 650,col=col[i], lwd=5)

text(730,y.max-.05-i*.02,paste0(“Rel = “,format(rel[i],nsmall=1)))



abline(h = .15,lty=2)

##################### THE END #################################

How Selection for Significance Influences Observed Power

Two years ago, I posted an Excel spreadsheet to help people to understand the concept of true power, observed power, and how selection for significance inflates observed power. Two years have gone by and I have learned R. It is time to update the post.

There is no mathematical formula to correct observed power for inflation to solve for true power. This was partially the reason why I created the R-Index, which is an index of true power, but not an estimate of true power.  This has led to some confusion and misinterpretation of the R-Index (Disjointed Thought blog post).

However, it is possible to predict median observed power given true power and selection for statistical significance.  To use this method for real data with observed median power of only significant results, one can simply generate a range of true power values, generate the predicted median observed power and then pick the true power value with the smallest discrepancy between median observed power and simulated inflated power estimates. This approach is essentially the same as the approach used by pcurve and puniform, which only
differ in the criterion that is being minimized.

Here is the r-code for the conversion of true.power into the predicted observed power after selection for significance.

true.power = seq(.01,.99,.01)
obs.pow = pnorm(qnorm(true.power/2+(1-true.power),qnorm(true.power,z.crit)),z.crit)

And here is a pretty picture of the relationship between true power and inflated observed power.  As we can see, there is more inflation for low true power because observed power after selection for significance has to be greater than 50%.  With alpha = .05 (two-tailed), when the null-hypothesis is true, inflated observed power is 61%.   Thus, an observed median power of 61% for only significant results supports the null-hypothesis.  With true power of 50%, observed power is inflated to 75%.  For high true power, the inflation is relatively small. With the recommended true power of 80%, median observed power for only significant results is 86%.


Observed power is easy to calculate from reported test statistics. The first step is to compute the exact two-tailed p-value.  These p-values can then be converted into observed power estimates using the standard normal distribution.

z.crit = qnorm(.975)
Obs.power = pnorm(qnorm(1-p/2),z.crit)

If there is selection for significance, you can use the previous formula to convert this observed power estimate into an estimate of true power.

This method assumes that (a) significant results are representative of the distribution and there are no additional biases (no p-hacking) and (b) all studies have the same or similar power.  This method does not work for heterogeneous sets of studies.

P.S.  It is possible to proof the formula that transforms true power into median observed power.  Another way to verify that the formula is correct is to confirm the predicted values with a simulation study.

Here is the code to run the simulation study:

n.sim = 100000
z.crit = qnorm(.975)
true.power = seq(.01,.99,.01)
obs.pow.sim = c()
for (i in 1:length(true.power)) {
z.sim = rnorm(n.sim,qnorm(true.power[i],z.crit))
med.z.sig = median(z.sim[z.sim > z.crit])
obs.pow.sim = c(obs.pow.sim,pnorm(med.z.sig,z.crit))

obs.pow = pnorm(qnorm(true.power/2+(1-true.power),qnorm(true.power,z.crit)),z.crit)



Reconstruction of a Train Wreck: How Priming Research Went off the Rails

Authors:  Ulrich Schimmack, Moritz Heene, and Kamini Kesavan


We computed the R-Index for studies cited in Chapter 4 of Kahneman’s book “Thinking Fast and Slow.” This chapter focuses on priming studies, starting with John Bargh’s study that led to Kahneman’s open email.  The results are eye-opening and jaw-dropping.  The chapter cites 12 articles and 11 of the 12 articles have an R-Index below 50.  The combined analysis of 31 studies reported in the 12 articles shows 100% significant results with average (median) observed power of 57% and an inflation rate of 43%.  The R-Index is 14. This result confirms Kahneman’s prediction that priming research is a train wreck and readers of his book “Thinking Fast and Slow” should not consider the presented studies as scientific evidence that subtle cues in their environment can have strong effects on their behavior outside their awareness.


In 2011, Nobel Laureate Daniel Kahneman published a popular book, “Thinking Fast and Slow”, about important finding in social psychology.

In the same year, questions about the trustworthiness of social psychology were raised.  A Dutch social psychologist had fabricated data. Eventually over 50 of his articles would be retracted.  Another social psychologist published results that appeared to demonstrate the ability to foresee random future events (Bem, 2011). Few researchers believed these results and statistical analysis suggested that the results were not trustworthy (Francis, 2012; Schimmack, 2012).  Psychologists started to openly question the credibility of published results.

In the beginning of 2012, Doyen and colleagues published a failure to replicate a prominent study by John Bargh that was featured in Daniel Kahneman’s book.  A few month later, Daniel Kahneman distanced himself from Bargh’s research in an open email addressed to John Bargh (Young, 2012):

“As all of you know, of course, questions have been raised about the robustness of priming results…. your field is now the poster child for doubts about the integrity of psychological research… people have now attached a question mark to the field, and it is your responsibility to remove it… all I have personally at stake is that I recently wrote a book that emphasizes priming research as a new approach to the study of associative memory…Count me as a general believer… My reason for writing this letter is that I see a train wreck looming.”

Five years later, Kahneman’s concerns have been largely confirmed. Major studies in social priming research have failed to replicate and the replicability of results in social psychology is estimated to be only 25% (OSC, 2015).

Looking back, it is difficult to understand the uncritical acceptance of social priming as a fact.  In “Thinking Fast and Slow” Kahneman wrote “disbelief is not an option. The results are not made up, nor are they statistical flukes. You have no choice but to accept that the major conclusions of these studies are true.”

Yet, Kahneman could have seen the train wreck coming. In 1971, he co-authored an article about scientists’ “exaggerated confidence in the validity of conclusions based on small samples” (Tversky & Kahneman, 1971, p. 105).  Yet, many of the studies described in Kahneman’s book had small samples.  For example, Bargh’s priming study used only 30 undergraduate students to demonstrate the effect.

Replicability Index

Small samples can be sufficient to detect large effects. However, small effects require large samples.  The probability of replicating a published finding is a function of sample size and effect size.  The Replicability Index (R-Index) makes it possible to use information from published results to predict how replicable published results are.

Every reported test-statistic can be converted into an estimate of power, called observed power. For a single study, this estimate is useless because it is not very precise. However, for sets of studies, the estimate becomes more precise.  If we have 10 studies and the average power is 55%, we would expect approximately 5 to 6 studies with significant results and 4 to 5 studies with non-significant results.

If we observe 100% significant results with an average power of 55%, it is likely that studies with non-significant results are missing (Schimmack, 2012).  There are too many significant results.  This is especially true because average power is also inflated when researchers report only significant results. Consequently, the true power is even lower than average observed power.  If we observe 100% significant results with 55% average powered power, power is likely to be less than 50%.

This is unacceptable. Tversky and Kahneman (1971) wrote “we refuse to believe that a serious investigator will knowingly accept a .50 risk of failing to confirm a valid research hypothesis.”

To correct for the inflation in power, the R-Index uses the inflation rate. For example, if all studies are significant and average power is 75%, the inflation rate is 25% points.  The R-Index subtracts the inflation rate from average power.  So, with 100% significant results and average observed power of 75%, the R-Index is 50% (75% – 25% = 50%).  The R-Index is not a direct estimate of true power. It is actually a conservative estimate of true power if the R-Index is below 50%.  Thus, an R-Index below 50% suggests that a significant result was obtained only by capitalizing on chance, although it is difficult to quantify by how much.

How Replicable are the Social Priming Studies in “Thinking Fast and Slow”?

Chapter 4: The Associative Machine

4.1.  Cognitive priming effect

In the 1980s, psychologists discovered that exposure to a word causes immediate and measurable changes in the ease with which many related words can be evoked.

[no reference provided]

4.2.  Priming of behavior without awareness

Another major advance in our understanding of memory was the discovery that priming is not restricted to concepts and words. You cannot know this from conscious experience, of course, but you must accept the alien idea that your actions and your emotions can be primed by events of which you are not even aware.

“In an experiment that became an instant classic, the psychologist John Bargh and his collaborators asked students at New York University—most aged eighteen to twenty-two—to assemble four-word sentences from a set of five words (for example, “finds he it yellow instantly”). For one group of students, half the scrambled sentences contained words associated with the elderly, such as Florida, forgetful, bald, gray, or wrinkle. When they had completed that task, the young participants were sent out to do another experiment in an office down the hall. That short walk was what the experiment was about. The researchers unobtrusively measured the time it took people to get from one end of the corridor to the other.”

“As Bargh had predicted, the young people who had fashioned a sentence from words with an elderly theme walked down the hallway significantly more slowly than the others. walking slowly, which is associated with old age.”

“All this happens without any awareness. When they were questioned afterward, none of the students reported noticing that the words had had a common theme, and they all insisted that nothing they did after the first experiment could have been influenced by the words they had encountered. The idea of old age had not come to their conscious awareness, but their actions had changed nevertheless.“

[John A. Bargh, Mark Chen, and Lara Burrows, “Automaticity of Social Behavior: Direct Effects of Trait Construct and Stereotype Activation on Action,” Journal of Personality and Social Psychology 71 (1996): 230–44.]

t(28)=2.86 0.008 2.66 0.76
t(28)=2.16 0.039 2.06 0.54

MOP = .65, Inflation = .35, R-Index = .30

4.3.  Reversed priming: Behavior primes cognitions

“The ideomotor link also works in reverse. A study conducted in a German university was the mirror image of the early experiment that Bargh and his colleagues had carried out in New York.”

“Students were asked to walk around a room for 5 minutes at a rate of 30 steps per minute, which was about one-third their normal pace. After this brief experience, the participants were much quicker to recognize words related to old age, such as forgetful, old, and lonely.”

“Reciprocal priming effects tend to produce a coherent reaction: if you were primed to think of old age, you would tend to act old, and acting old would reinforce the thought of old age.”

t(18)=2.10 0.050 1.96 0.50
t(35)=2.10 0.043 2.02 0.53
t(31)=2.50 0.018 2.37 0.66

MOP = .53, Inflation = .47, R-Index = .06

4.4.  Facial-feedback hypothesis (smiling makes you happy)

“Reciprocal links are common in the associative network. For example, being amused tends to make you smile, and smiling tends to make you feel amused….”

“College students were asked to rate the humor of cartoons from Gary Larson’s The Far Side while holding a pencil in their mouth. Those who were “smiling” (without any awareness of doing so) found the cartoons funnier than did those who were “frowning.”

[“Inhibiting and Facilitating Conditions of the Human Smile: A Nonobtrusive Test of the Facial Feedback Hypothesis,” Journal of Personality and Social Psychology 54 (1988): 768–77.]

The authors used the more liberal and unconventional criterion of p < .05 (one-tailed), z = 1.65, as a criterion for significance. Accordingly, we adjusted the R-Index analysis and used 1.65 as the criterion value.

t(89)=1.85 0.034 1.83 0.57
t(75)=1.78 0.034 1.83 0.57

MOP = .57, Inflation = .43, R-Index = .14

These results could not be replicated in a large replication effort with 17 independent labs. Not a single lab produced a significant result and even a combined analysis failed to show any evidence for the effect.

4.5. Automatic Facial Responses

In another experiment, people whose face was shaped into a frown (by squeezing their eyebrows together) reported an enhanced emotional response to upsetting pictures—starving children, people arguing, maimed accident victims.

[Ulf Dimberg, Monika Thunberg, and Sara Grunedal, “Facial Reactions to

Emotional Stimuli: Automatically Controlled Emotional Responses,” Cognition and Emotion, 16 (2002): 449–71.]

The description in the book does not match any of the three studies reported in this article. The first two studies examined facial muscle movements in response to pictures of facial expressions (smiling or frowning faces).  The third study used emotional pictures of snakes and flowers. We might consider the snake pictures as being equivalent to pictures of starving children or maimed accident victims.  Participants were also asked to frown or to smile while looking at the pictures. However, the dependent variable was not how they felt in response to pictures of snakes, but rather how their facial muscles changed.  Aside from a strong effect of instructions, the study also found that the emotional picture had an automatic effect on facial muscles.  Participants frowned more when instructed to frown and looking at a snake picture than when instructed to frown and looking at a picture of a flower. “This response, however, was larger to snakes than to flowers as indicated by both the Stimulus factor, F(1, 47) = 6.66, p < .02, and the Stimulus 6 Interval factor, F(1, 47) = 4.30, p < .05.”  (p. 463). The evidence for smiling was stronger. “The zygomatic major muscle response was larger to flowers than to snakes, which was indicated by both the Stimulus factor, F(1, 47) = 18.03, p < .001, and the Stimulus 6 Interval factor, F(1, 47) = 16.78, p < .001.”  No measures of subjective experiences were included in this study.  Therefore, the results of this study provide no evidence for Kahneman’s claim in the book and the results of this study are not included in our analysis.

4.6.  Effects of Head-Movements on Persuasion

“Simple, common gestures can also unconsciously influence our thoughts and feelings.”

“In one demonstration, people were asked to listen to messages through new headphones. They were told that the purpose of the experiment was to test the quality of the audio equipment and were instructed to move their heads repeatedly to check for any distortions of sound. Half the participants were told to nod their head up and down while others were told to shake it side to side. The messages they heard were radio editorials.”

“Those who nodded (a yes gesture) tended to accept the message they heard, but those who shook their head tended to reject it. Again, there was no awareness, just a habitual connection between an attitude of rejection or acceptance and its common physical expression.”

F(2,66)=44.70 0.000 7.22 1.00

MOP = 1.00, Inflation = .00,  R-Index = 1.00

[Gary L. Wells and Richard E. Petty, “The Effects of Overt Head Movements on Persuasion: Compatibility and Incompatibility of Responses,” Basic and Applied Social Psychology, 1, (1980): 219–30.]

4.7   Location as Prime

“Our vote should not be affected by the location of the polling station, for example, but it is.”

“A study of voting patterns in precincts of Arizona in 2000 showed that the support for propositions to increase the funding of schools was significantly greater when the polling station was in a school than when it was in a nearby location.”

“A separate experiment showed that exposing people to images of classrooms and school lockers also increased the tendency of participants to support a school initiative. The effect of the images was larger than the difference between parents and other voters!”

[Jonah Berger, Marc Meredith, and S. Christian Wheeler, “Contextual Priming: Where People Vote Affects How They Vote,” PNAS 105 (2008): 8846–49.]

z = 2.10 0.036 2.10 0.56
p = .05 0.050 1.96 0.50

MOP = .53, Inflation = .47, R-Index = .06

4.8  Money Priming

“Reminders of money produce some troubling effects.”

“Participants in one experiment were shown a list of five words from which they were required to construct a four-word phrase that had a money theme (“high a salary desk paying” became “a high-paying salary”).”

“Other primes were much more subtle, including the presence of an irrelevant money-related object in the background, such as a stack of Monopoly money on a table, or a computer with a screen saver of dollar bills floating in water.”

“Money-primed people become more independent than they would be without the associative trigger. They persevered almost twice as long in trying to solve a very difficult problem before they asked the experimenter for help, a crisp demonstration of increased self-reliance.”

“Money-primed people are also more selfish: they were much less willing to spend time helping another student who pretended to be confused about an experimental task. When an experimenter clumsily dropped a bunch of pencils on the floor, the participants with money (unconsciously) on their mind picked up fewer pencils.”

“In another experiment in the series, participants were told that they would shortly have a get-acquainted conversation with another person and were asked to set up two chairs while the experimenter left to retrieve that person. Participants primed by money chose to stay much farther apart than their nonprimed peers (118 vs. 80 centimeters).”

“Money-primed undergraduates also showed a greater preference for being alone.”

[Kathleen D. Vohs, “The Psychological Consequences of Money,” Science 314 (2006): 1154–56.]

F(2,49)=3.73 0.031 2.16 0.58
t(35)=2.03 0.050 1.96 0.50
t(37)=2.06 0.046 1.99 0.51
t(42)=2.13 0.039 2.06 0.54
F(2,32)=4.34 0.021 2.30 0.63
t(38)=2.13 0.040 2.06 0.54
t(33)=2.37 0.024 2.26 0.62
F(2,58)=4.04 0.023 2.28 0.62
chi^2(2)=10.10 0.006 2.73 0.78

MOP = .58, Inflation = .42, R-Index = .16

4.9  Death Priming

“The evidence of priming studies suggests that reminding people of their mortality increases the appeal of authoritarian ideas, which may become reassuring in the context of the terror of death.”

The cited article does not directly examine this question.  The abstract states that “three experiments were conducted to test the hypothesis, derived from terror management theory, that reminding people of their mortality increases attraction to those who consensually validate their beliefs and decreases attraction to those who threaten their beliefs” (p. 308).  Study 2 found no general effect of death priming. Rather, the effect was qualified by authoritarianism. Mortality salience enhanced the rejection of dissimilar others in Study 2 only among high authoritarian subjects.” (p. 314), based on a three-way interaction with F(1,145) = 4.08, p = .045.  We used the three-way interaction for the computation of the R-Index.  Study 1 reported opposite effects for ratings of Christian targets, t(44) = 2.18, p = .034 and Jewish targets, t(44)= 2.08, p = .043. As these tests are dependent, only one test could be used, and we chose the slightly stronger result.  Similarly, Study 3 reported significantly more liking of a positive interviewee and less liking of a negative interviewee, t(51) = 2.02, p = .049 and t(49) = 2.42, p = .019, respectively. We chose the stronger effect.

[Jeff Greenberg et al., “Evidence for Terror Management Theory II: The Effect of Mortality Salience on Reactions to Those Who Threaten or Bolster the Cultural Worldview,” Journal of Personality and Social Psychology]

t(44)=2.18 0.035 2.11 0.56
F(1,145)=4.08 0.045 2.00 0.52
t(49)=2.42 0.019 2.34 0.65

MOP = .56, Inflation = .44, R-Index = .12

4.10  The “Lacy Macbeth Effect”

“For example, consider the ambiguous word fragments W_ _ H and S_ _ P. People who were recently asked to think of an action of which they are ashamed are more likely to complete those fragments as WASH and SOAP and less likely to see WISH and SOUP.”

“Furthermore, merely thinking about stabbing a coworker in the back leaves people more inclined to buy soap, disinfectant, or detergent than batteries, juice, or candy bars. Feeling that one’s soul is stained appears to trigger a desire to cleanse one’s body, an impulse that has been dubbed the “Lady Macbeth effect.”

[Lady Macbeth effect”: Chen-Bo Zhong and Katie Liljenquist, “Washing Away Your Sins:

Threatened Morality and Physical Cleansing,” Science 313 (2006): 1451–52.]

F(1,58)=4.26 0.044 2.02 0.52
F(1,25)=6.99 0.014 2.46 0.69

MOP = .61, Inflation = .39, R-Index = .22

The article reports two more studies that are not explicitly mentioned, but are used as empirical support for the Lady Macbeth effect. As the results of these studies were similar to those in the mentioned studies, including these tests in our analysis does not alter the conclusions.

chi^2(1)=4.57 0.033 2.14 0.57
chi^2(1)=5.02 0.025 2.24 0.61

MOP = .59, Inflation = .41, R-Index = .18

4.11  Modality Specificity of the “Lacy Macbeth Effect”

“Participants in an experiment were induced to “lie” to an imaginary person, either on the phone or in e-mail. In a subsequent test of the desirability of various products, people who had lied on the phone preferred mouthwash over soap, and those who had lied in e-mail preferred soap to mouthwash.”

[Spike Lee and Norbert Schwarz, “Dirty Hands and Dirty Mouths: Embodiment of the Moral-Purity Metaphor Is Specific to the Motor Modality Involved in Moral Transgression,” Psychological Science 21 (2010): 1423–25.]

The results are presented as significant with a one-sided t-test. “As shown in Figure 1a, participants evaluated mouthwash more positively after lying in a voice mail (M = 0.21, SD = 0.72) than after lying in an e-mail (M = –0.26, SD = 0.94), F(1, 81) = 2.93, p = .03 (one-tailed), d = 0.55 (simple main effect), but evaluated hand sanitizer more positively after lying in an e-mail (M = 0.31, SD = 0.76) than after lying in a voice mail (M = –0.12, SD = 0.86), F(1, 81) = 3.25, p = .04 (one-tailed), d = 0.53 (simple main effect).”  We adjusted the significance criterion for the R-Index accordingly.

F(1,81)=2.93 0.045 1.69 0.52
F(1,81)=3.25 0.038 1.78 0.55

MOP = .54, Inflation = .46, R-Index = .08

4.12   Eyes on You

“On the first week of the experiment (which you can see at the bottom of the figure), two wide-open eyes stare at the coffee or tea drinkers, whose average contribution was 70 pence per liter of milk. On week 2, the poster shows flowers and average contributions drop to about 15 pence. The trend continues. On average, the users of the kitchen contributed almost three times as much in ’eye weeks’ as they did in ’flower weeks.’ ”

[Melissa Bateson, Daniel Nettle, and Gilbert Roberts, “Cues of Being Watched Enhance Cooperation in a Real-World Setting,” Biology Letters 2 (2006): 412–14.]

F(1,7)=11.55 0.011 2.53 0.72

MOP = .72, Inflation = .28, R-Index = .44

Combined Analysis

We then combined the results from the 31 studies mentioned above.  While the R-Index for small sets of studies may underestimate replicability, the R-Index for a large set of studies is more accurate.  Median Obesrved Power for all 31 studies is only 57%. It is incredible that 31 studies with 57% power could produce 100% significant results (Schimmack, 2012). Thus, there is strong evidence that the studies provide an overly optimistic image of the robustness of social priming effects.  Moreover, median observed power overestimates true power if studies were selected to be significant. After correcting for inflation, the R-Index is well below 50%.  This suggests that the studies have low replicability. Moreover, it is possible that some of the reported results are actually false positive results.  Just like the large-scale replication of the facial feedback studies failed to provide any support for the original findings, other studies may fail to show any effects in large replication projects. As a result, readers of “Thinking Fast and Slow” should be skeptical about the reported results and they should disregard Kahneman’s statement that “you have no choice but to accept that the major conclusions of these studies are true.”  Our analysis actually leads to the opposite conclusion. “You should not accept any of the conclusions of these studies as true.”

k = 31,  MOP = .57, Inflation = .43, R-Index = .14,  Grade: F for Fail

Powergraph of Chapter 4kfs

Schimmack and Brunner (2015) developed an alternative method for the estimation of replicability.  This method takes into account that power can vary across studies. It also provides 95% confidence intervals for the replicability estimate.  The results of this method are presented in the Figure above. The replicability estimate is similar to the R-Index, with 14% replicability.  However, due to the small set of studies, the 95% confidence interval is wide and includes values above 50%. This does not mean that we can trust the published results, but it does suggest that some of the published results might be replicable in larger replication studies with more power to detect small effects.  At the same time, the graph shows clear evidence for a selection effect.  That is, published studies in these articles do not provide a representative picture of all the studies that were conducted.  The powergraph shows that there should have been a lot more non-significant results than were reported in the published articles.  The selective reporting of studies that worked is at the core of the replicability crisis in social psychology (Sterling, 1959, Sterling et al., 1995; Schimmack, 2012).  To clean up their act and to regain trust in published results, social psychologists have to conduct studies with larger samples that have more than 50% power (Tversky & Kahneman, 1971) and they have to stop reporting only significant results.  We can only hope that social psychologists will learn from the train wreck of social priming research and improve their research practices.

Are Most Published Results in Psychology False? An Empirical Study

Why Most Published Research Findings  are False by John P. A. Ioannidis

In 2005, John P. A. Ioannidis wrote an influential article with the title “Why Most Published Research Findings are False.” The article starts with the observation that “there is increasing concern that most current published research findings are false” (e124). Later on, however, the concern becomes a fact. “It can be proven that most claimed research findings are false” (e124). It is not surprising that an article that claims to have proof for such a stunning claim has received a lot of attention (2,199 citations and 399 citations in 2016 alone in Web of Science).

Most citing articles focus on the possibility that many or even more than half of all published results could be false. Few articles cite Ioannidis to make the factual statement that most published results are false, and there appears to be no critical examination of Ioannidis’s simulations that he used to support his claim.

This blog post shows that these simulations make questionable assumptions and shows with empirical data that Ioannidis’s simulations are inconsistent with actual data.

Critical Examination of Ioannidis’s Simulations

First, it is important to define what a false finding is. In many sciences, a finding is published when a statistical test produced a significant result (p < .05). For example, a drug trial may show a significant difference between a drug and a placebo control condition with a p-value of .02. This finding is then interpreted as evidence for the effectiveness of the drug.

How could this published finding be false? The logic of significance testing makes this clear. The only inference that is being made is that the population effect size (i.e., the effect size that could be obtained if the same experiment were repeated with an infinite number of participants) is different from zero and in the same direction as the one observed in the study. Thus, the claim that most significant results are false implies that in more than 50% of all published significant results the null-hypothesis was true. That is, a false positive result was reported.

Ioannidis then introduces the positive predictive value (PPV). The positive predictive value is the proportion of positive results (p < .05) that are true positives.

(1) PPV = TP/(TP + FP)

PTP = True Positive Results, FP = False Positive Results

The proportion of true positive results (TP) depends on the percentage of true hypothesis (PTH) and the probability of producing a significant result when a hypothesis is true. This probability is known as statistical power. Statistical power is typically defined as 1 minus the type-II error (beta).

(2) TP = PTH * Power = PTH * (1 – beta)

The probability of a false positive result depends on the proportion of false hypotheses (PFH) and the criterion for significance (alpha).

(3) FP = PFH * alpha

This means that the actual proportion of true significant results is a function of the ratio of true and false hypotheses (PTH:PFH), power, and alpha.

(4) PPV = (PTH*power) / ((PTH*power) + (PFH * alpha))

Ioannidis translates his claim that most published findings are false into a PPV below 50%. This would mean that the null-hypothesis is true in more than 50% of published results that falsely rejected it.

(5) (PTH*power) / ((PTH*power) + (PFH * alpha))  < .50

Equation (5) can be simplied to the inequality equation

(6) alpha > PTH/PFH * power

We can rearrange formula (6) and substitute PFH with (1-PHT) to determine the maximum proportion of true hypotheses to produce over 50% false positive results.

(7a)  =  alpha = PTH/(1-PTH) * power

(7b) = alpha*(1-PTH) = PTH * power

(7c) = alpha – PTH*alpha = PTH * power

(7d) =  alpha = PTH*alpha + PTH*power

(7e) = alpha = PTH(alpha + power)

(7f) =  alpha/(power + alpha) = PTH


Table 1 shows the results.

Power                  PTH / PFH             
90%                       5  / 95
80%                       6  / 94
70%                       7  / 93
60%                       8  / 92
50%                       9  / 91
40%                      11 / 89
30%                       14 / 86
20%                      20 / 80
10%                       33 / 67                     

Even if researchers would conduct studies with only 20% power to discover true positive results, we would only obtain more than 50% false positive results if only 20% of hypothesis were true. This makes it rather implausible that most published results could be false.

To justify his bold claim, Ioannidis introduces the notion of bias. Bias can be introduced due to various questionable research practices that help researchers to report significant results. The main effect of these practices is that the probability of a false positive result to become significant increases.

Simmons et al. (2011) showed that massive use several questionable research practices (p-hacking) can increase the risk of a false positive result from the nominal 5% to 60%. If we assume that bias is rampant and substitute the nominal alpha of 5% with an assumed alpha of 50%, fewer false hypotheses are needed to produce more false than true positives (Table 2).

Power                 PTH/PFH             
90%                     40 / 60
80%                     43 / 57
70%                     46 / 54
60%                     50 / 50
50%                     55 / 45
40%                     60 / 40
30%                     67 / 33
20%                     75 / 25
10%                      86 / 14                    

If we assume that bias inflates the risk of type-I errors from 5% to 60%, it is no longer implausible that most research findings are false. In fact, more than 50% of published results would be false if researchers tested hypothesis with 50% power and 50% of tested hypothesis are false.

However, the calculations in Table 2 ignore the fact that questionable research practices that inflate false positives also decrease the rate of false negatives. For example, a researcher who continues testing until a significant result is obtained, increases the chances of obtaining a significant result no matter whether the hypothesis is true or false.

Ioannidis recognizes this, but he assumes that bias has the same effect for true hypothesis and false hypothesis. This assumption is questionable because it is easier to produce a significant result if an effect exists than if no effect exists. Ioannidis’s assumption implies that bias increases the proportion of false positive results a lot more than the proportion of true positive results.

For example, if power is 50%, only 50% of true hypothesis produce a significant result. However, with a bias factor of .4, another 40% of the false negative results will become significant, adding another .4*.5 = 20% true positive results to the number of true positive results. This gives a total of 70% positive results, which is a 40% increase over the number of positive results that would have been obtained without bias. However, this increase in true positive results pales in comparison to the effect that 40% bias has on the rate of false positives. As there are 95% true negatives, 40% bias produces another .95*.40 = 38% of false positive results. So instead of 5% false positive results, bias increases the percentage of false positive results from 5% to 43%, an increase by 760%. Thus, the effect of bias on the PPV is not equal. A 40% increase of false positives has a much stronger impact on the PPV than a 40% increase of true positives. Ioannidis provides no rational for this bias model.

A bigger concern is that Ioannidis makes sweeping claims about the proportion of false published findings based on untested assumptions about the proportion of null-effects, statistical power, and the amount of bias due to questionable research practices.
For example, he suggests that 4 out of 5 discoveries in adequately powered (80% power) exploratory epidemiological studies are false positives (PPV = .20). To arrive at this estimate, he assumes that only 1 out of 11 hypotheses is true and that for every 1000 studies, bias adds only 1000* .30*.10*.20 = 6 true positives results compared to 1000* .30*.90*.95 = 265 false positive results (i.e., 44:1 ratio). The assumed bias turns a PPV of 62% without bias into a PPV of 20% with bias. These untested assumptions are used to support the claim that “simulations show that for most study designs and settings, it is more likely for a research claim to be false than true.” (e124).

Many of these assumptions can be challenged. For example, statisticians have pointed out that the null-hypothesis is unlikely to be true in most studies (Cohen, 1994). This does not mean that all published results are true, but Ioannidis’ claims rest on the opposite assumption that most hypothesis are a priori false. This makes little sense when the a priori hypothesis is specified as a null-effect and even a small effect size is sufficient for a hypothesis to be correct.

Ioannidis also ignores attempts to estimate the typical power of studies (Cohen, 1962). At least in psychology, the typical power is estimated to be around 50%. As shown in Table 2, even massive bias would still produce more true than false positive results, if the null-hypothesis is false in no more than 50% of all statistical tests.

In conclusion, Ioannidis’s claim that most published results are false depends heavily on untested assumptions and cannot be considered a factual assessment of the actual number of false results in published journals.

Testing Ioannidis’s Simulations

10 years after the publication of “Why Most Published Research Findings Are False,”  it is possible to put Ioannidis’s simulations to an empirical test. Powergraphs (Schimmack, 2015) can be used to estimate the average replicability of published test results. For this purpose, each test statistic is converted into a z-value. A powergraph is foremost a histogram of z-values. The distribution of z-values provides information about the average statistical power of published results because studies with higher power produce higher z-values.

Figure 1 illustrates the distribution of z-values that is expected for Ioanndis’s model for “adequately powered exploratory epidemiological study” (Simulation 6 in Figure 4). Ioannidis assumes that for every true positive, there are 10 false positives (R = 1:10). He also assumed that studies have 80% power to detect a true positive. In addition, he assumed 30% bias.


A 30% bias implies that for every 100 false hypotheses, there would be 33 (100*[.30*.95+.05]) rather than 5 false positive results (.95*.30+.05)/.95). The effect on false negatives is much smaller (100*[.30*.20 + .80]). Bias was modeled by increasing the number of attempts to produce a significant result so that proportion of true and false hypothesis matched the predicted proportions. Given an assumed 1:10 ratio of true to false hypothesis, the ratio is 335 false hypotheses to 86 true hypotheses. The simulation assumed that researchers tested 100,000 false hypotheses and observed 35000 false positive results and that they tested 10,000 true hypotheses and observed 8,600 true positive results. Bias was simulated by increasing the number of tests to produce the predicted ratio of true and false positive results.

Figure 1 only shows significant results because only significant results would be reported as positive results. Figure 1 shows that a high proportion of z-values are in the range between 1.95 (p = .05) and 3 (p = .001). Powergraphs use z-curve (Schimmack & Brunner, 2016) to estimate the probability that an exact replication study would replicate a significant result. In this simulation, this probability is a mixture of false positives and studies with 80% power. The true average probability is 20%. The z-curve estimate is 21%. Z-curve can also estimate the replicability for other sets of studies. The figure on the right shows replicability for studies that produced an observed z-score greater than 3 (p < .001). The estimate shows an average replicability of 59%. Thus, researchers can increase the chance of replicating published findings by adjusting the criterion value and ignoring significant results with p-values greater than p = .001, even if they were reported as significant with p < .05.

Figure 2 shows the distribution of z-values for Ioannidis’s example of a research program that produces more true than false positives, PPV = .85 (Simulation 1 in Table 4).


Visual inspection of Figure 1 and Figure 2 is sufficient to show that a robust research program produces a dramatically different distribution of z-values. The distribution of z-values in Figure 2 and a replicability estimate of 67% are impossible if most of the published significant results were false.  The maximum value that could be obtained is obtained with a PPV of 50% and 100% power for the true positive results, which yields a replicability estimate of .05*.50 + 1*.50 = 55%. As power is much lower than 100%, the real maximum value is below 50%.

The powergraph on the right shows the replicability estimate for tests that produced a z-value greater than 3 (p < .001). As only a small proportion of false positives are included in this set, z-curve correctly estimates the average power of these studies as 80%. These examples demonstrate that it is possible to test Ioannidis’s claim that most published (significant) results are false empirically. The distribution of test results provides relevant information about the proportion of false positives and power. If actual data are more similar to the distribution in Figure 1, it is possible that most published results are false positives, although it is impossible to distinguish false positives from false negatives with extremely low power. In contrast, if data look more like those in Figure 2, the evidence would contradict Ioannidis’s bold and unsupported claim that most published results are false.

The maximum replicabiltiy that could be obtained with 50% false-positives would require that the true positive studies have 100% power. In this case, replicability would be .50*.05 + .50*1 = 52.5%.  However, 100% power is unrealistic. Figure 3 shows the distribution for a scenario with 90% power and 100% bias and an equal percentage of true and false hypotheses. The true replicabilty for this scenario is .05*.50 + .90 * .50 = 47.5%. z-curve slightly overestimates replicabilty and produced an estimate of 51%.  Even 90% power is unlikely in a real set of data. Thus, replicability estimates above 50% are inconsistent with Ioannidis’s hypothesis that most published positive results are false.  Moreover, the distribution of z-values greater than 3 is also informative. If positive results are a mixture of many false positive results and true positive results with high power, the replicabilty estimate for z-values greater than 3 should be high. In contrast, if this estimate is not much higher than the estimate for all z-values, it suggest that there is a high proportion of studies that produced true positive results with low power.


Empirical Evidence

I have produced powergraphs and replicability estimates for over 100 psychology journals (2015 Replicabilty Rankings). Not a single journal produced a replicability estimate below 50%. Below are a few selected examples.

The Journal of Experimental Psychology: Learning, Memory and Cognition publishes results from cognitive psychology. In 2015, a replication project (OSC, 2015) demonstrated that 50% of significant results produced a significant result in a replication study. It is unlikely that all non-significant results were false positives. Thus, the results show that Ioannidis’s claim that most published results are false does not apply to results published in this journal.

Powergraphs for JEP-LMC3.g

The powergraphs further support this conclusion. The graphs look a lot more like Figure 2 than Figure 1 and the replicability estimate is even higher than the one expected from Ioannidis’s simulation with a PPV of 85%.

Another journal that was subjected to replication attempts was Psychological Science. The success rate for Psychological Science was below 50%. However, it is important to keep in mind that a non-significant result in a replication study does not prove that the original result was a false positive. Thus, the PPV could still be greater than 50%.

Powergraphs for PsySci3.g

The powergraph for Psychological Science shows more z-values in the range between 2 and 3 (p > .001). Nevertheless, the replicability estimate is comparable to the one in Figure 2 which simulated a high PPV of 85%. Closer inspection of the results published in this journal would be required to determine whether a PPV below .50 is plausible.

The third journal that was subjected to a replication attempt was the Journal of Personality and Social Psychology. The journal has three sections, but I focus on the Attitude and Social Cognition section because many replication studies were from this section. The success rate of replication studies was only 25%. However, there is controversy about the reason for this high number of failed replications and once more it is not clear what percentage of failed replications were due to false positive results in the original studies.

Powergraphs for JPSP-ASC3.g

One problem with the journal rankings is that they are based on automated extraction of all test results. Ioannidis might argue that his claim focused only on test results that tested an original, novel, or an important finding, whereas articles also often report significance tests for other effects. For example, an intervention study may show a strong decrease in depression, when only the interaction with treatment is theoretically relevant.

I am currently working on powergraphs that are limited to theoretically important statistical tests. These results may show lower replicability estimates. Thus, it remains to be seen how consistent Ioannidis’s predictions are for tests of novel and original hypotheses. Powergraphs provide a valuable tool to address this important question.

Moreover, powergraphs can be used to examine whether science is improving. So far, powergraphs of psychology journals have shown no systematic improvement in response to concerns about high false positive rates in published journals. The powergraphs for 2016 will be published soon. Stay tuned.


Reexamining Cunningham, Preacher, and Banaji’s Multi-Method Model of Racism Measures

William A. Cunningham, Kristopher J. Preacher, and Mahzarin R. Banaji. (2001).
Implicit Attitude Measures: Consistency, Stability, and Convergent Validity, Psychological Science, 12(2), 163-170.

In recent years, several techniques have been developed to measure implicit social cognition. Despite their increased use, little attention has been devoted to their reliability and validity. This article undertakes a direct assessment of the interitem consistency, stability, and convergent validity of some implicit attitude measures. Attitudes toward blacks and whites were measured on four separate occasions, each 2 weeks apart, using three relatively implicit measures (response window evaluative priming, the Implicit Association Test, and the response-window Implicit Association Test) and one explicit measure (Modern Racism Scale). After correcting for interitem inconsistency with latent variable analyses, we found that (a) stability indices improved and (b) implicit measures were substantially correlated with each other, forming a single latent factor. The psychometric properties of response-latency implicit measures have greater integrity than recently suggested.

Critique of Original Article

This article has been cited 362 times (Web of Science, January 2017).  It still is one of the most rigorous evaluations of the psychometric properties of the race Implicit Association Test (IAT).  As noted in the abstract, the strength of the study is the use of several implicit measures and the repeated measurement of attitudes on four separate occasions.  This design makes it possible to separate several variance components in the race IAT.  First, it is possible to examine how much variance is explained by causal factors that are stable over time and shared by implicit and explicit attitude measures.  Second, it is possible to measure the amount of variance that is unique to the IAT.  As this component is not shared with other implicit measures, this variance can be attributed to systematic measurement error that is stable over time.  A third variance component is variance that is shared only with other implicit measures and that is stable over time. This variance component could reflect stable implicit racial attitudes.  Finally, it is possible to identify occasion specific variance in attitudes.  This component would reveal systematic changes in implicit attitudes.

The original article presents a structural equation model that makes it possible to identify some of these variance components.  However, the model is not ideal for this purpose and the authors do not test some of these variance components.  For example, the model does not include any occasion specific variation in attitudes.  This could be because attitudes do not vary over the one-month interval of the study, or it could mean that the model failed to specify this variance component.

This reanalysis also challenges the claim by the original authors that they provided evidence for a dissociation of implicit and explicit attitudes.  “We found a dissociation between implicit and explicit measures of race attitude: Participants simultaneously self-reported nonprejudiced explicit attitudes toward black Americans while showing an implicit difficulty in associating black with positive attributes” (p. 169). The main problem is that the design does not allow to make this claim because the study included only a single explicit racism measure.  Consequently, it is impossible to determine whether unique variance in the explicit measure reflects systematic measurement in explicit attitude measures (social desirable responding, acquiescence response styles) or whether this variance reflects consciously accessible attitudes that are distinct from implicit attitudes.  In this regard, the authors claim that “a single-factor solution does not fit the data” (p. 170) is inconsistent with their own structural equation model that shows a single second-order factor that explains the covariance among the three implicit measures and the explicit measure.

The authors caution that a single IAT measure is not very reliable, but their statement about reliability is vague. “Our analyses of implicit attitude measures suggest that the degree of measurement error in response-latency measures can be substantial; estimates of Cronbach’s alpha indicated that, on average, more than 30% of the variance associated with the measurements was random error.” (p. 160).  More than 30% random measurement error leaves a rather large range of reliability estimates ranging from 0% to 70%.   The respective parameter estimates for the IAT in Figure 4 are .53^2 = .28, .65^2 = .42, .74^2 = .55, and .38^2 = .14.  These reliability estimates vary considerably due to the small sample size, but the loading of the first IAT would suggest that only 19% of the variance in a single IAT is reliable. As reliablity is the upper limit for validity, it would imply that no more than 20% of the variance in a single IAT captures variation in implicit racial attitudes.

The authors caution readers about the use of a single IAT to measure implicit attitudes. “When using latency-based measures as indices of individual differences, it may be essential to employ analytic techniques, such as covariance structure modeling, that can separate measurement error from a measure of individual differences. Without such analyses, estimates of relationships involving implicit measures may produce misleading null results” (p. 169).  However, the authors fail to mention that the low reliability of a single IAT also has important implications for the use of the IAT for the assessment of implicit prejudice.  Given this low estimate of validity, users of the Harvard website that provides information about individual’s performance on the IAT should be warned that the feedback is neither reliable nor valid by conventional standards for psychological tests.

Reanalysis of Published Correlation Matrix

The Table below reproduces the correlation matrix. The standard deviations in the last row are rescaled to avoid rounding problems. This has no effect on the results.

.80   1
.78 .82  1
.76 .77 .86   1
.21 .15 .15 .14   1
.13 .14 .10 .08 .31  1
.16 .26 .23 .20 .42 .50 1
.14 .17 .16 .13 .16 .33 .17 1
.20 .16 .19 .26 .33 .11 .23 .07 1
.26 .29 .18 .19 .20 .27 .36 .29 .26   1
.35 .33 .34 .25 .28 .29 .34 .33 .36 .39   1
.19 .17 .08 .07 .12 .25 .30 .14 .01 .17 .24 1
.00 .11 .07 .04 .27 .18 .19 .02 .03 .01 .02 .07 1
.16 .08 .04 .08 .26 .27 .24 .22 .14 .32 .32 .17 .13 1
.12 .01 .02 .07 .13 .19 .18 .00 .02 .00 .11 .04 .17 .30 1
.33 .18 .26 .31 .14 .24 .31 .15 .22 .20 .27 .04 .01 .48 .42 1

SD 0.84 0.82 0.88 0.86 2.2066 1.2951 1.0130 0.9076 1.2 1.0 1.1 1.0 0.7 0.8 0.8 0.9

1-4 = Modern Racism Scale (1-4); 5-8 Implicit Association Test (1-4);  9-12 = Response Window IAT (1-4);  13-16 Response Window Evaluative Priming (1-4)


Fitting the data to the original model reproduced the original results.  I then fitted the data to a model with a single attitude factor (see Figure 1).  The model also allowed for measure-specific variances.  An initial model showed no significant measure-specific variances for the two versions of the IAT .  Hence, these method factors were not included in the final model.  To control for variance that is clearly consciously accessible, I modeled the relationship between the explicit factor and the attitude factor as a causal path from the explicit factor to the attitude factor.  This path should not be interpreted as a causal relationship in this case. Rather the path can be used to estimate how much of the variance in the attitude factor is explained by consciously accessible information that influences the explicit measure.  In this model, the residual variance is variation that is shared among implicit measures, but not with the explicit measure.

The model had good fit to the data.  I then imposed constraints on factor loadings.  The constrained model had better fit than the unconstrained model (delta AIC = 4.60, delta BIC = 43.53).  The main finding is that the standard IAT had a loading of .55 on the attitude factor.  The indirect path from the implicit attitude factor to a single IAT measure is only slightly smaller, .55*.92 = .51.  The 95%CI for this parameter ranged from .41 to .60.  The upper bound of the 95%CI would imply that at most 36% of the variance in a single IAT reflects implicit racial attitudes.  However, it is important to note that the model in Figure 1 assumes that the Modern Racism Scale is a perfectly valid measure of consciously accessible attitudes. Any systematic measurement error in the Modern Racism Scale would reduce the amount of variance in the attitude factor that reflects unconscious factors.  Again, the lack of multiple explicit measures makes it impossible to separate systematic measurement error from valid variance in explicit measures.  Thus, the amount of variance in a single IAT that reflects unconscious racial attitudes can range from 0 to 36%.

How Variable are Implicit Racial Attitudes?

The design repeated measurement of implicit attitudes on four occasions.  If recent experiences influence implicit attitudes, we would expect that implicit measures of attitudes on the same occasion are more highly correlated with each other than implicit measures taken on different occasions.  Given the low validity of implicit attitude measures, I examined this question with constrained parameters. By estimating a single parameter, the model has more power to reveal a consistent relationship between implicit measures that were obtained during the same testing session.  Neither the two IATs, nor the IAT and the evaluative priming task (EP) showed significant occasion-specific variance.  Although this finding may be due to low power to detect occasion specific variation, this finding suggests that most of the variance in an IAT is due to stable variation and random measurement error.


Cunningham et al. (2001) conducted a rigorous psychometric study of the Implicit Association Test.  The original article reported results that could be reproduced.  The authors correctly interpret their results as evidence that a single IAT has low reliability. However, they falsely imply that their results provide evidence that the IAT and other implicit measures are valid measures of an implicit form of racism that is not consciously accessible.  My new analysis shows that their results are consistent with this hypothesis, if one assumes that the Modern Racism Scale is a perfectly valid measure of consciously accessible racial attitudes.  Under this assumption, about 25% (95%CI 16-36) of the variance in a single IAT would reflect implicit attitudes.  However, it is rather unlikely that the Modern Racism Scale is a perfect measure of explicit racial attitudes, and the amount of variance in performance on the IAT that reflects unconscious racism is likely to be smaller. Another important finding that was implicit, but not explicitly mentioned, in the original model is that there is no evidence for situation-specific variation in implicit attitudes. At least over the one-month period of the study, racial attitudes remained stable and did not vary as a function of naturally occurring events that might influence racial attitudes (e.g., positive or negative intergroup contact).  This finding may explain why experimental manipulations of implicit attitudes also often produce very small effects (Joy Gaba & Nosek, 2010).

One surprising finding was that the IAT showed no systematic measurement error in this model. This would imply that repeated measures of the IAT could be used to measure racial attitudes with high validity.  Unfortunately, most studies with the IAT rely on a single testing situation and ignore that most of the variance in a single IAT is measurement error.  To improve research on racial attitudes and prejudice, social psychologists should use multiple explicit and implicit measures and use structural equation models to examine which variance components of a measurement model of racial attitudes predict actual behavior.

Validity of the Implicit Association Test as a Measure of Implicit Attitudes

This blog post reports the results of an analysis of correlations among 4 explicit and 3 implicit attitude measures published by Ranganath, Tucker, and Nosek (2008).

Original article:
Kate A. Ranganath, Colin Tucker Smith, & Brian A. Nosek (2008). Distinguishing automatic and controlled components of attitudes from direct and indirect measurement methods. Journal of Experimental Social Psychology 44 (2008) 386–396; doi:10.1016/j.jesp.2006.12.008

Distinct automatic and controlled processes are presumed to influence social evaluation. Most empirical approaches examine automatic processes using indirect methods, and controlled processes using direct methods. We distinguished processes from measurement methods to test whether a process distinction is more useful than a measurement distinction for taxonomies of attitudes. Results from two studies suggest that automatic components of attitudes can be measured directly. Direct measures of automatic attitudes were reports of gut reactions (Study 1) and behavioral performance in a speeded self-report task (Study 2). Confirmatory factor analyses comparing two factor models revealed better fits when self-reports of gut reactions and speeded self-reports shared a factor with automatic measures versus sharing a factor with controlled self-report measures. Thus, distinguishing attitudes by the processes they are presumed to measure (automatic versus controlled) is more meaningful than distinguishing based on the directness of measurement.

Description of Original Study

Study 1 measured relative attitudes towards heterosexuals and homosexuals with seven measures; four explicit measures and three reaction time tasks. Specifically, the four explicit measures were

Actual = Participants were asked to report their “actual feelings” towards gay and straight people when given enough time for full consideration on a scale ranging from 1=very negative to 8 = very positive.

Gut = Participants were asked to report their “gut reaction” towards gay and straight people when given enough time for full consideration on a scale ranging from 1=very negative to 8 = very positive.

Time0 and Time5: A second explicit rating task assessed an “attitude timeline”. Participants reported their attitudes toward the two groups at multiple time points: (1) instant reaction, (2) reaction a split-second later, (3) reaction after 1 s, (4) reaction after 5 s, and (5) reaction when given enough time to think fully. Only the first (Time0) and the last (Time5) rating were included in the model.

The three reaction time measures were the Implicit Association Test (IAT), the Go-NoGo Association Test (GNAT), and a Four-Category Sorting Paired Features Task (SPF). All three measures use differences in response times to measure attitudes.

Table A1 in the Appendix reported the correlations among the seven tasks.

GNAT .36 1
SPF .26 .18 1
GUT .23 .33 .12 1
Actual .16 .31 .01 .65 1
Time0 .19 .31 .16 .85 .50 1
Time5 .01 .24 .01 .54 .81 .50 1

The authors tested a variety of structural equation models. The best fitting model, preferred by the authors, was a model with three correlated latent factors. “In this three-factor model, self-reported gut feelings (GutFeeling, Instant Feeling) comprised their own attitude factor distinct from a factor comprised of the indirect, automatic measures (IAT, GNAT, SPF) and from a factor comprised of the direct, controlled measures (Actual Feeling, Fully Considered Feeling). The data were an excellent fit (chi^2(12) = 10.8).

The authors then state “while self-reported gut feelings were more similar to the indirect measures than to the other self-reported attitude measures, there was some unique variance in self-reported gut feelings that was distinct from both.” (p. 391) and they go on to speculate that “one possibility is that these reports are a self-theory that has some but not complete correspondence with automatic evaluations” (p. 391). The also consider the possibility that “measures like the IAT, GNAT, and SPF partly assess automatic evaluations that are “experienced” and amenable to introspective report, and partly evaluations that are not” (p. 391). But they favor the hypothesis that “self-report of ‘gut feelings’ is a meaningful account of some components of automatic evaluation” (p. 391). The interpret these results as strong support for their “contention that a taxonomy of attitudes by measurement features is not as effective as one that distinguishes by presumed component processes” (p. 391). The conclusion reiterates this point. “The present studies suggest that attitudes have distinct but related automatic and controlled factors contributing to social evaluation and that parsing attitudes by underlying processes is superior to parsing attitude measures by measurement features” (p. 393). Surprisingly, the author do not mention the three-factor model in the Discussion and rather claim support for a two-factor model that distinguishes processes rather than measures (explicit vs. implicit). “In both studies, model comparison using confimatory factor analysis showed the data were better fit to a two-factor model distinguishing automatic and controlled components of attitudes than to a model distinguishing attitudes by whether they were measured directly or indirectly” (p. 393). The authors then suggest that some explicit measures (ratings of gut reactions) can measure automatic attitudes. “These findings suggest that direct measures can be devised to capture automatic components of attitudes despite suggestions that indirect measures are essential for such assessments” (p. 393).

New Analysis 

The main problem with this article is that the author never report parameter estimates for the model. Depending on the pattern of correlations among the three factors and factor loadings, the interpretation of the results can change. I first tried to fit the three-factor model to the covariance matrix (setting variances to 1) to the published correlation matrix. MPLUS7.1 showed some problems with negative residual variance for Actual. Also the model had one less degree of freedom than the published model. However, fixing the residual variance of actual did not solve the problem. I then proceeded to fit my own model. The model is essentially the same model as the three-factor model with the exception that I modeled the correlation among the three-latent factor with a single higher-order factor. This factor represents variation in common causes that influences all attitude measures. The problem of negative variance in the actual measure was solved by allowing for an extra correlation between the actual and gut ratings. As seen in the correlation table, these two explicit measures correlated more highly with each other (r = .65) than the corresponding T0 and T5 measures (rs = .54, .50). As in the original article, model fit was good (see Figure). Figure 1 shows for the first time the parameter estimates of the model.



The loadings of the explicit measures on the primary latent factors are above .80. For single item measures, this implies that these ratings are essentially measuring the same construct with some random error. Thus, the latent factors can be interpreted as explicit ratings of affective responses immediately or after some reflection. The loadings of these two factors on the higher order factor show that reflective and immediate responses are strongly influenced by the common factor. This is not surprising. Reflection may alter the immediate response somewhat, but it is unlikely to reverse or dramatically change the response a few seconds later. Interestingly, the immediate response has a higher loading on the attitude factor, although in this small sample the differences in loadings is not significant (chi^2(1) = 0.22. The third primary factor represents the shared variance among the three reaction time measures. It also loads on the general attitude factor, but the loading is weaker than the loading for the explicit measures. The parameter estimates suggest that about 25% of the variance is explained by the common attitude (.51^2) and 75% is unique to the reaction time measures. This variance component can be interpreted as unique variance in implicit measures. The factor loadings of the three reaction time measures are also relevant. The loading of the IAT suggests that only 28% (.53^2) of the observed variance in the IAT reflects the effect of causal factors that influence reaction time measures of attitudes. As some of this variance is also shared with explicit measures, only 21% ((.86*.53)^2) of the variance in the IAT represents the variance in the implicit attitude factor This has important implications for the use of the IAT to examine potential effects of implicit attitudes on behavior. Even if implicit attitudes had a strong effect on a behavior (r = .5), the correlation between IAT scores and the behavior only would be r = .86*.53*.5 = .23. A sample size of N = 146 participants would be needed to have 80% power to provide significant evidence for such a relationship (p < .05, two-tailed). Given a more modest effect of attitudes on behavior, r = .86*.53*.30 = .14, the sample size would need to be larger (N = 398). As many studies of implicit attitudes and behavior used smaller samples, we would expect many non-significant results, unless non-significant results remain unreported and published results report inflated effect sizes. One solution to the problem of low power in studies of implicit attitudes would be the use of multiple implicit attitude measures. This study suggests that a battery of different reaction time tasks can be used to remove random and task specific measurement error. Such a multi-method approach to the measurement of implicit attitudes is highly recommended for future studies because it would also help to interpret results of studies in which implicit attitudes do not influence behavior. If a set of implicit measures show convergent validity, this finding would indicate that implicit attitudes did not influence the behavior. In contrast, a null-result with a single implicit measure may simply show that the measure failed to measure implicit attitudes.


This article reported some interesting data, but failed to report the actual results. This analysis of the data showed that explicit measures are highly correlated with each other and show discriminant validity from implicit, reaction time measures. The new analysis also made it possible to estimate the amount of variance in the Implicit Association Test that reflects variance that is not shared with explicit measures but shared with other implicit measures. The estimate of 20% suggests that most of the variance in the IAT is due to factors other than implicit attitudes and that the test cannot be used to diagnose individuals. Whether the 20% of variance that is uniquely shared with other implicit measures reflects unconscious attitudes or method variance that is common to reaction time tasks remains unclear. The model also implies that predictive validity of a single IAT for prejudice behaviors is expected to be small to moderate (r < .30), which means large samples are needed to study the effect of implicit attitudes on behavior.